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Yes, this is exactly what I observed And once the "New ergosteral " is dead or not allowed to grow, the algae then becomes enticing and edible to the cleanup crew as well.The genus Bryopsidales (bryopsis, derbesia, caulerpa, ulva, codium, halimeda) are algae with a cenocytic cell structure, do not have complete cell division and all algae functions as a single cell. Fluconazole blocks the synthesis of "new ergosterol," but does not remove ergosterol that is already on the cell wall. The alga has to grow (form more wall) so that, in the absence of ergosterol, this "new wall" is fragile and allows the environment to penetrate the cell and disorganize it ... so bryopsis "begins to die" by the growth tips and the disorganization is "top-down" in the filaments, until it becomes complete.
Strong hug
Wouldn't removal of the larger algal masses allow more of the medication to be available to the remainder?Yes!
You would be pruning the "sick part" and favoring the recovery of the seaweed, which delays the process.
Strong hug!
I found that doing that a few days before treating with fluconazole that would be true however not during treatment. Because it is absorbed through the newest tips/growth of the algae. On my experiment I pulled off two sections after treatment they were the last two actually die off even after shaded areas died.Wouldn't removal of the larger algal masses allow more of the medication to be available to the remainder?
Yes, but once the plant is trimmed , it leaves small tips, the only point of contact with the water, those points should try to grow.I found that doing that a few days before treating with fluconazole that would be true however not during treatment. Because it is absorbed through the newest tips/growth of the algae. On my experiment I pulled off two sections after treatment they were the last two actually die off even after shaded areas died.
Maybe it has something to do with the species of bryopsis, the lighting (as we do know that well lit areas die more quickly) ... it's hard to say for sure. I'm also thinking that it also depends on your individual tanks chemistry. As we know all tanks are not the same. Some died much faster than others. Yours was gone within 5 days but mine took double that.Yes, but once the plant is trimmed , it leaves small tips, the only point of contact with the water, those points should try to grow.
In the first tank i treated , I needed my toothbrush/comfort blankie, and hit the high spots. None of it came back and was by eye bryo free in less then five days. In the second ,larger tank, I only hit a few of the high spots , those didn't come back and the unpruned patches took longer to die.
Thus the very interesting question of , scrub or not to scrubMaybe it has something to do with the species of bryopsis, the lighting (as we do know that well lit areas die more quickly) ... it's hard to say for sure. I'm also thinking that it also depends on your individual tanks chemistry. As we know all tanks are not the same. Some died much faster than others. Yours was gone within 5 days but mine took double that.
LOL for sure! However since we're not sure we could try the easy way, dose the tank and wait and see. If it comes back then we know that that particular tank needs to be scrubbed first then retreated.Thus the very interesting question of , scrub or not to scrub
The problem is how the medicine has to act to do its job; Fluconazole will only interfere with the new walls that are forming, in the growth shoots, it is from there that the disorganization of the algae begins.Wouldn't removal of the larger algal masses allow more of the medication to be available to the remainder?
I do wonder why, when I put this theory into practice, it worked better to scrub it than to leave it.The problem is how the medicine has to act to do its job; Fluconazole will only interfere with the new walls that are forming, in the growth shoots, it is from there that the disorganization of the algae begins.
If we cut diseased sprouts, the alga has a chance to "coagulate" its wounds, as if it had been bitten by a tang or snail, and sprout from the roots.
Remaining diseased shoots, as the cenocitic algae do not possess true cell division in their tissues, the imbalance walks from the tips down, and compromises to the roots.
I think the way to speed up the destruction of algae in this particular mode of action of Fluconazole is to accelerate its growth, perhaps with adjustment of the spectrum and PAR of the luminaires, besides the accumulation of nitrate and phosphate by the removal of the cup of the skimmer, which also favors.
Strong hug
Makes sense. They do smell rotting food.[CITA = "prsnlty, post: 4208609, member: 17049"] Sim, isso é exatamente o que eu observei : DE uma vez que o "New ergosteral" está morto ou não está autorizado a crescer, as algas então se tornam atraentes e comestíveis para a equipe de limpeza bem. [/ QUOTE]
De alguma forma, os caminhos de produção de alleloquímicos que defendem algas da herbivoria também são comprometidos pelo Fluconazol. Na verdade, quando a alga começa a sofrer e mesmo antes do clareamento, as espigas e os caracóis já se aproximam e começam a morder, o que não antes.
Abraço forte
I may be wrong, I just suppose that wayI do wonder why, when I put this theory into practice, it worked better to scrub it than to leave it.
"I think the way to speed up the destruction of algae in this particular mode of action of Fluconazole is to accelerate its growth, perhaps with adjustment of the spectrum and PAR of the luminaires, besides the accumulation of nitrate and phosphate by the removal of the cup of the skimmer, which also favors."Somehow, the pathways of production of allelochemicals that defend algae from herbivory, are also compromised by Fluconazole. In fact, when the alga begins to suffer and even before the whitening, the tangs and snails already approach and begin to bite, which they did not before.
Strong hug
The problem is how the medicine has to act to do its job; Fluconazole will only interfere with the new walls that are forming, in the growth shoots, it is from there that the disorganization of the algae begins.
If we cut diseased sprouts, the alga has a chance to "coagulate" its wounds, as if it had been bitten by a tang or snail, and sprout from the roots.
Remaining diseased shoots, as the cenocitic algae do not possess true cell division in their tissues, the imbalance walks from the tips down, and compromises to the roots.
g
What evidence is there for that?